WEKO3
アイテム
{"_buckets": {"deposit": "a246ed1b-749e-4913-8977-d8aa297f8ec7"}, "_deposit": {"id": "3728", "owners": [], "pid": {"revision_id": 0, "type": "depid", "value": "3728"}, "status": "published"}, "_oai": {"id": "oai:soar-ir.repo.nii.ac.jp:00003728", "sets": ["462"]}, "author_link": ["6614", "6615", "6616"], "item_1628147817048": {"attribute_name": "出版タイプ", "attribute_value_mlt": [{"subitem_version_resource": "http://purl.org/coar/version/c_ab4af688f83e57aa", "subitem_version_type": "AM"}]}, "item_6_biblio_info_6": {"attribute_name": "書誌情報", "attribute_value_mlt": [{"bibliographicIssueDates": {"bibliographicIssueDate": "2014-03", "bibliographicIssueDateType": "Issued"}, "bibliographicIssueNumber": "2", "bibliographicPageEnd": "371", "bibliographicPageStart": "363", "bibliographicVolumeNumber": "51", "bibliographic_titles": [{"bibliographic_title": "VETERINARY PATHOLOGY"}]}]}, "item_6_description_20": {"attribute_name": "抄録", "attribute_value_mlt": [{"subitem_description": "Amyloidoses are a group of protein-misfolding disorders that are characterized by the deposition of amyloid fibrils in organs and/or tissues. In reactive amyloid A (AA) amyloidosis, serum AA (SAA) protein forms deposits in mice, domestic and wild animals, and humans that experience chronic inflammation. AA amyloid fibrils are abnormal beta-sheet-rich forms of the serum precursor SAA, with conformational changes that promote fibril formation. Extracellular deposition of amyloid fibrils causes disease in affected animals. Recent findings suggest that AA amyloidosis could be transmissible. Similar to the pathogenesis of transmissible prion diseases, amyloid fibrils induce a seeding-nucleation process that may lead to development of AA amyloidosis. We review studies of possible transmission in bovine, avian, mouse, and cheetah AA amyloidosis.", "subitem_description_type": "Abstract"}]}, "item_6_description_30": {"attribute_name": "資源タイプ(コンテンツの種類)", "attribute_value_mlt": [{"subitem_description": "Article", "subitem_description_type": "Other"}]}, "item_6_description_5": {"attribute_name": "引用", "attribute_value_mlt": [{"subitem_description": "VETERINARY PATHOLOGY. 51(2):363-371 (2014)", "subitem_description_type": "Other"}]}, "item_6_link_3": {"attribute_name": "信州大学研究者総覧へのリンク", "attribute_value_mlt": [{"subitem_link_text": "Higuchi, K", "subitem_link_url": "http://soar-rd.shinshu-u.ac.jp/profile/ja.gCfUPmAh.html"}]}, "item_6_link_67": {"attribute_name": "WoS", "attribute_value_mlt": [{"subitem_link_text": "Web of Science", "subitem_link_url": "http://gateway.isiknowledge.com/gateway/Gateway.cgi?\u0026GWVersion=2\u0026SrcAuth=ShinshuUniv\u0026SrcApp=ShinshuUniv\u0026DestLinkType=FullRecord\u0026DestApp=WOS\u0026KeyUT=000332610500006"}]}, "item_6_publisher_4": {"attribute_name": "出版者", "attribute_value_mlt": [{"subitem_publisher": "SAGE PUBLICATIONS INC"}]}, "item_6_relation_47": {"attribute_name": "PubMed", "attribute_value_mlt": [{"subitem_relation_name": [{"subitem_relation_name_text": "24280941"}], "subitem_relation_type_id": {"subitem_relation_type_id_text": "https://pubmed.ncbi.nlm.nih.gov/24280941", "subitem_relation_type_select": "PMID"}}]}, "item_6_relation_48": {"attribute_name": "DOI", "attribute_value_mlt": [{"subitem_relation_name": [{"subitem_relation_name_text": "10.1177/0300985813511128"}], "subitem_relation_type_id": {"subitem_relation_type_id_text": "https://doi.org/10.1177/0300985813511128", "subitem_relation_type_select": "DOI"}}]}, "item_6_rights_62": {"attribute_name": "権利", "attribute_value_mlt": [{"subitem_rights": "The final, definitive version of this paper has been published in Veterinary Pathology, vol. 51 no. 2, March 2014 by SAGE Publications Ltd, All rights reserved. Copyright© 2014 American College of Veterinary Pathologists"}]}, "item_6_select_64": {"attribute_name": "著者版フラグ", "attribute_value_mlt": [{"subitem_select_item": "author"}]}, "item_6_source_id_35": {"attribute_name": "ISSN", "attribute_value_mlt": [{"subitem_source_identifier": "0300-9858", "subitem_source_identifier_type": "PISSN"}]}, "item_6_source_id_39": {"attribute_name": "NII ISSN", "attribute_value_mlt": [{"subitem_source_identifier": "0300-9858", "subitem_source_identifier_type": "PISSN"}]}, "item_6_source_id_40": {"attribute_name": "書誌レコードID", "attribute_value_mlt": [{"subitem_source_identifier": "AA00883666", "subitem_source_identifier_type": "NCID"}]}, "item_6_text_69": {"attribute_name": "wosonly authkey", "attribute_value_mlt": [{"subitem_text_value": "prion-like transmission; cheetah; amyloid-enhancing factor; serum amyloid A; mouse; AA amyloidosis; bird; cattle"}]}, "item_6_text_70": {"attribute_name": "wosonly keywords", "attribute_value_mlt": [{"subitem_text_value": "MOUSE SENILE AMYLOIDOSIS; ENHANCING FACTOR; EXPERIMENTAL INDUCTION; AVIAN AMYLOIDOSIS; RENAL AMYLOIDOSIS; RAPID INDUCTION; FIBRIL PROTEIN; SERUM; CATTLE; MICE"}]}, "item_6_textarea_68": {"attribute_name": "wosonly abstract", "attribute_value_mlt": [{"subitem_textarea_value": "Amyloidoses are a group of protein-misfolding disorders that are characterized by the deposition of amyloid fibrils in organs and/or tissues. In reactive amyloid A (AA) amyloidosis, serum AA (SAA) protein forms deposits in mice, domestic and wild animals, and humans that experience chronic inflammation. AA amyloid fibrils are abnormal beta-sheet-rich forms of the serum precursor SAA, with conformational changes that promote fibril formation. Extracellular deposition of amyloid fibrils causes disease in affected animals. Recent findings suggest that AA amyloidosis could be transmissible. Similar to the pathogenesis of transmissible prion diseases, amyloid fibrils induce a seeding-nucleation process that may lead to development of AA amyloidosis. We review studies of possible transmission in bovine, avian, mouse, and cheetah AA amyloidosis."}]}, "item_creator": {"attribute_name": "著者", "attribute_type": "creator", "attribute_value_mlt": [{"creatorNames": [{"creatorName": "Murakami, T", "creatorNameLang": "en"}], "nameIdentifiers": [{"nameIdentifier": "6614", "nameIdentifierScheme": "WEKO"}]}, {"creatorNames": [{"creatorName": "Ishiguro, N", "creatorNameLang": "en"}], "nameIdentifiers": [{"nameIdentifier": "6615", "nameIdentifierScheme": "WEKO"}]}, {"creatorNames": [{"creatorName": "Higuchi, K", "creatorNameLang": "en"}], "nameIdentifiers": [{"nameIdentifier": "6616", "nameIdentifierScheme": "WEKO"}]}]}, "item_files": {"attribute_name": "ファイル情報", "attribute_type": "file", "attribute_value_mlt": [{"accessrole": "open_date", "date": [{"dateType": "Available", "dateValue": "2015-09-24"}], "displaytype": "detail", "download_preview_message": "", "file_order": 0, "filename": "Transmission_Systemic_AA_Amyloidosis_Animals.pdf", "filesize": [{"value": "571.8 kB"}], "format": "application/pdf", "future_date_message": "", "is_thumbnail": false, "licensetype": "license_note", "mimetype": "application/pdf", "size": 571800.0, "url": {"label": "Transmission_Systemic_AA_Amyloidosis_Animals.pdf", "url": "https://soar-ir.repo.nii.ac.jp/record/3728/files/Transmission_Systemic_AA_Amyloidosis_Animals.pdf"}, "version_id": "c0eb7e70-c353-4ce4-90fc-2f22d1027787"}]}, "item_keyword": {"attribute_name": "キーワード", "attribute_value_mlt": [{"subitem_subject": "prion-like transmission", "subitem_subject_scheme": "Other"}, {"subitem_subject": "cheetah", "subitem_subject_scheme": "Other"}, {"subitem_subject": "amyloid-enhancing factor", "subitem_subject_scheme": "Other"}, {"subitem_subject": "serum amyloid A", "subitem_subject_scheme": "Other"}, {"subitem_subject": "mouse", "subitem_subject_scheme": "Other"}, {"subitem_subject": "AA amyloidosis", "subitem_subject_scheme": "Other"}, {"subitem_subject": "bird", "subitem_subject_scheme": "Other"}, {"subitem_subject": "cattle", "subitem_subject_scheme": "Other"}]}, "item_language": {"attribute_name": "言語", "attribute_value_mlt": [{"subitem_language": "eng"}]}, "item_resource_type": {"attribute_name": "資源タイプ", "attribute_value_mlt": [{"resourcetype": "journal article", "resourceuri": "http://purl.org/coar/resource_type/c_6501"}]}, "item_title": "Transmission of Systemic AA Amyloidosis in Animals", "item_titles": {"attribute_name": "タイトル", "attribute_value_mlt": [{"subitem_title": "Transmission of Systemic AA Amyloidosis in Animals", "subitem_title_language": "en"}]}, "item_type_id": "6", "owner": "1", "path": ["462"], "permalink_uri": "http://hdl.handle.net/10091/17606", "pubdate": {"attribute_name": "PubDate", "attribute_value": "2014-06-17"}, "publish_date": "2014-06-17", "publish_status": "0", "recid": "3728", "relation": {}, "relation_version_is_last": true, "title": ["Transmission of Systemic AA Amyloidosis in Animals"], "weko_shared_id": -1}
Transmission of Systemic AA Amyloidosis in Animals
http://hdl.handle.net/10091/17606
http://hdl.handle.net/10091/1760636ade4cc-50e1-4705-bb58-9278cc24136f
名前 / ファイル | ライセンス | アクション |
---|---|---|
Transmission_Systemic_AA_Amyloidosis_Animals.pdf (571.8 kB)
|
|
Item type | 学術雑誌論文 / Journal Article(1) | |||||
---|---|---|---|---|---|---|
公開日 | 2014-06-17 | |||||
タイトル | ||||||
言語 | en | |||||
タイトル | Transmission of Systemic AA Amyloidosis in Animals | |||||
言語 | ||||||
言語 | eng | |||||
DOI | ||||||
識別子タイプ | DOI | |||||
関連識別子 | https://doi.org/10.1177/0300985813511128 | |||||
関連名称 | 10.1177/0300985813511128 | |||||
キーワード | ||||||
主題 | prion-like transmission, cheetah, amyloid-enhancing factor, serum amyloid A, mouse, AA amyloidosis, bird, cattle | |||||
資源タイプ | ||||||
資源 | http://purl.org/coar/resource_type/c_6501 | |||||
タイプ | journal article | |||||
著者 |
Murakami, T
× Murakami, T× Ishiguro, N× Higuchi, K |
|||||
信州大学研究者総覧へのリンク | ||||||
氏名 | Higuchi, K | |||||
URL | http://soar-rd.shinshu-u.ac.jp/profile/ja.gCfUPmAh.html | |||||
出版者 | ||||||
出版者 | SAGE PUBLICATIONS INC | |||||
引用 | ||||||
内容記述タイプ | Other | |||||
内容記述 | VETERINARY PATHOLOGY. 51(2):363-371 (2014) | |||||
書誌情報 |
VETERINARY PATHOLOGY 巻 51, 号 2, p. 363-371, 発行日 2014-03 |
|||||
抄録 | ||||||
内容記述タイプ | Abstract | |||||
内容記述 | Amyloidoses are a group of protein-misfolding disorders that are characterized by the deposition of amyloid fibrils in organs and/or tissues. In reactive amyloid A (AA) amyloidosis, serum AA (SAA) protein forms deposits in mice, domestic and wild animals, and humans that experience chronic inflammation. AA amyloid fibrils are abnormal beta-sheet-rich forms of the serum precursor SAA, with conformational changes that promote fibril formation. Extracellular deposition of amyloid fibrils causes disease in affected animals. Recent findings suggest that AA amyloidosis could be transmissible. Similar to the pathogenesis of transmissible prion diseases, amyloid fibrils induce a seeding-nucleation process that may lead to development of AA amyloidosis. We review studies of possible transmission in bovine, avian, mouse, and cheetah AA amyloidosis. | |||||
資源タイプ(コンテンツの種類) | ||||||
内容記述タイプ | Other | |||||
内容記述 | Article | |||||
ISSN | ||||||
収録物識別子タイプ | PISSN | |||||
収録物識別子 | 0300-9858 | |||||
書誌レコードID | ||||||
収録物識別子タイプ | NCID | |||||
収録物識別子 | AA00883666 | |||||
PubMed | ||||||
識別子タイプ | PMID | |||||
関連識別子 | https://pubmed.ncbi.nlm.nih.gov/24280941 | |||||
関連名称 | 24280941 | |||||
権利 | ||||||
権利情報 | The final, definitive version of this paper has been published in Veterinary Pathology, vol. 51 no. 2, March 2014 by SAGE Publications Ltd, All rights reserved. Copyright© 2014 American College of Veterinary Pathologists | |||||
出版タイプ | ||||||
出版タイプ | AM | |||||
出版タイプResource | http://purl.org/coar/version/c_ab4af688f83e57aa | |||||
WoS | ||||||
表示名 | Web of Science | |||||
URL | http://gateway.isiknowledge.com/gateway/Gateway.cgi?&GWVersion=2&SrcAuth=ShinshuUniv&SrcApp=ShinshuUniv&DestLinkType=FullRecord&DestApp=WOS&KeyUT=000332610500006 |