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Myeloid progenitors with PTPN11 and nonRAS pathway gene mutations are refractory to treatment with 6-mercaptopurine in juvenile myelomonocytic leukemia
http://hdl.handle.net/10091/17717
http://hdl.handle.net/10091/177178f0a4b1b-eed1-4ba0-b3dc-a10fabbb4ba6
名前 / ファイル | ライセンス | アクション |
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Item type | 学術雑誌論文 / Journal Article(1) | |||||
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公開日 | 2014-07-14 | |||||
タイトル | ||||||
言語 | en | |||||
タイトル | Myeloid progenitors with PTPN11 and nonRAS pathway gene mutations are refractory to treatment with 6-mercaptopurine in juvenile myelomonocytic leukemia | |||||
言語 | ||||||
言語 | eng | |||||
キーワード | ||||||
主題Scheme | Other | |||||
主題 | PTPN11 | |||||
キーワード | ||||||
主題Scheme | Other | |||||
主題 | SETBP1 | |||||
キーワード | ||||||
主題Scheme | Other | |||||
主題 | JAK3 | |||||
キーワード | ||||||
主題Scheme | Other | |||||
主題 | 6-MP | |||||
キーワード | ||||||
主題Scheme | Other | |||||
主題 | JMML | |||||
資源タイプ | ||||||
資源 | http://purl.org/coar/resource_type/c_6501 | |||||
タイプ | journal article | |||||
著者 |
Matsuda, K
× Matsuda, K× Nakazawa, Y× Iwashita, C× Kurata, T× Hirabayashi, K× Saito, S× Tanaka, M× Yoshikawa, K× Yanagisawa, R× Sakashita, K× Sasaki, S× Honda, T× Koike, K |
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信州大学研究者総覧へのリンク | ||||||
氏名 | Nakazawa, Y | |||||
URL | https://soar-rd.shinshu-u.ac.jp/profile/ja.yCSFuNkh.html | |||||
信州大学研究者総覧へのリンク | ||||||
氏名 | Honda, T | |||||
URL | https://soar-rd.shinshu-u.ac.jp/profile/ja.WeDUHFkh.html | |||||
出版者 | ||||||
出版者 | NATURE PUBLISHING GROUP | |||||
引用 | ||||||
内容記述タイプ | Other | |||||
内容記述 | LEUKEMIA. 28(7):1545-1548 (2014) | |||||
書誌情報 |
LEUKEMIA 巻 28, 号 7, p. 1545-1548, 発行日 2014-07 |
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内容記述 | ||||||
内容記述タイプ | Other | |||||
内容記述 | advance online publication, February 25, 2014 | |||||
抄録 | ||||||
内容記述タイプ | Abstract | |||||
内容記述 | Juvenile myelomonocytic leukemia (JMML) is a fatal, mixed myeloproliferative and myelodysplastic disorder occurring in infancy and early childhood. Children with JMML have mutually exclusive genetic abnormalities in granulocyte-macrophage colony-stimulating factor (GM-CSF) signaling pathways, inactivation of the NF1 or mutations in PTPN11, NRAS, KRAS and CBL. A whole-exome sequencing study, performed by Sakaguchi et al.,3 has recently demonstrated that in addition to the high frequency of RAS pathway mutations, mutations in SETBP1 and JAK3 are common recurrent secondary events, and that these events may be involved in tumor progression, and are associated with poor clinical outcomes. | |||||
資源タイプ(コンテンツの種類) | ||||||
内容記述タイプ | Other | |||||
内容記述 | Article | |||||
ISSN | ||||||
収録物識別子タイプ | PISSN | |||||
収録物識別子 | 0887-6924 | |||||
書誌レコードID | ||||||
収録物識別子タイプ | NCID | |||||
収録物識別子 | AA12305962 | |||||
PubMed | ||||||
識別子タイプ | PMID | |||||
関連識別子 | https://pubmed.ncbi.nlm.nih.gov/24496301 | |||||
関連名称 | 24496301 | |||||
DOI | ||||||
識別子タイプ | DOI | |||||
関連識別子 | https://doi.org/10.1038/leu.2014.58 | |||||
関連名称 | 10.1038/leu.2014.58 | |||||
出版タイプ | ||||||
出版タイプ | AM | |||||
出版タイプResource | http://purl.org/coar/version/c_ab4af688f83e57aa | |||||
WoS | ||||||
表示名 | Web of Science | |||||
URL | http://gateway.isiknowledge.com/gateway/Gateway.cgi?&GWVersion=2&SrcAuth=ShinshuUniv&SrcApp=ShinshuUniv&DestLinkType=FullRecord&DestApp=WOS&KeyUT=000339705500022 |